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Click photo to enlarge Hands and feet
Birth Defects (In
Detail) An
injured hand is often the dominant hand of the only bread-earner of a
family and the incapacitation it causes not only effects the livelihood
of the patient but the well being of the entire family. It is possible
to examine, document and analyze hand functions, or the lack of it, but
the art is in learning how each patient uses these various attributes
in his daily life, his occupation, his hobbies and his leisure activities.
The challenge is not only to heal the injured hand but to get the person
back to his or her profession and give them back their hobbies and simple
pleasure pursuits. Now if the demand is that big why are we still trivializing
hand injuries and not giving it the respect and recognition it deserves? In
the course of training for General Surgery the special requirements of
the Hand seldom receive emphasis even in post-graduate spheres. Hand Injury
therefore has traditionally been a Surgical Cinderella, the responsibility
of many but the concern of a very few. Again as most patients in the developing
world who suffer Hand Injuries belong to the lower socioeconomic strata
and are without an insurance back up, this surgery, unlike say Cardiac
Surgery, has not attracted publicity, sponsorships and glamour. While
a lot of school going children aspire to become Cosmetic Surgeons or Cardiac
Surgeons not many of them dream to become Hand Surgeons! It is this image
which requires a change and various Hand Societies both National and International,
have taken up this image building project in a big way. The twelve commandments of management of Hand trauma are:
A
fully developed Hand Surgery service is manned by a multi-disciplinary
team comprising of a surgeon, a physician, a physiotherapist, an occupational
therapist, a splint technician, a psychologist and social workers. The
surgeon being the coordinator of the team is responsible for diagnosis
and surgery. These are however the first two steps in the ladder of recovery.
Close cooperation and constant inputs from the other members of the team
at appropriate time ensures a complete and uneventful recovery.
No treatment programme is complete without addressing the aspect of prevention. Hand injuries are invariably preventable – whether sustained at home from a faulty electrical appliance, or at work from a poorly designed thresher, or in the factories from carelessness and inexperience. Work exhaustion, fatigue, tobacco and other addictions, irregular supply of electricity are factors which catches one unaware and it is usually one’s hands that pay the price. REPLANTS
AND REVASCULARIZATIONS Replantation of limbs and digits are now a reality but the doctors in the periphery need to be educated regarding the storage and transportation of the amputated parts and the patient. All dismembered parts of the body should be put in a clean polythene bag, which in turn should be put in a flask or an icebox containing ice or yet another polythene bag filled with ice. The part should not come in direct contact with ice. It should now be transported by the fastest available conveyance to a Hand Surgery centre, even if the patient is being held back for more pressing emergencies like head or chest injuries. The doctor attending the patient in the emergency should not use his own discretion on whether the amputated parts are replantable or not but should send all the dismembered parts. Even if they are not transplantable in their original location they can serve as valuable sources of auto-graft for other locations. Thus a toe or a vascularised composite graft containing skin, muscle, fascia, bone, tendon and nerve in various combinations can be harvested from a crushed and useless lower limb for transplantation to an equally mutilated upper limb, which thus salvaged will function better than the best available prosthesis.
Not all amputated parts can be replanted. The perils of replantation surgery are:
Proximal
level of amputation
·
Vessel repair is easy ·
Permissible ischemia time is less ·
Peripheral nerves are mixed nerves Avulsed
nature of amputation
·
Vessels avulsed – extensive intimal damage ·
Nerves avulsed – damage to vasa nervosum ·
Tendons avulsed from myo-tendenous jn. ·
Horner’s Syndrome in higher injuries Cooling
before revascularization avoids
·
Oedema of amputated part ·
Increased Potassium levels ·
Increased metabolic acidosis ( lactates ) Incomplete
amputations
·
Splintage – prevent kinking / injury to vessels · Artery to be tied - no unsupported artery forceps or tourniquet should be used. Irreversibly damaged partsThese
must be transported with the patient as they form invaluable source of
skin and fascial flaps and artery, vein, nerve, tendon and bone
graft. We have even utilized a great toe and a second toe from an
irecoverably damaged below knee amputation part to provide the patient a
thumb and a digit to his injured upper limb Preservation and Transpotration Part should not
Transportation
POST
BURN CONTRANTURES OF HAND Post Burn Contractures of the hand involving the dorsum, flexor aspect of the fingers and thumb and the first web space are a crippling disability. They in varying combinations render the hand totally incapable of performing basic functions of pinch, hook and grasp. Avoiding them in the early nursing phase of Burns by proper splintage and timely escharectomy and skin grafting followed by physiotherapy remains the best treatment. Once formed, release of these contractures and cover by local, distant or free flaps and subsequent physiotherapy improves the functional capability of these hands considerably. 172
Post Burn Contracture hands in 164 individuals were surgically treated and
156 of them have returned to their previous vocation. The remaining 8 had
to opt for a change in vocation after proper training. Presently all
patients are gainfully employed. ADDUCTION CONTRACTURE THUMB Adduction contracture of the thumb or 1st. web space contracture effects predominantly the movements of the trapezio-metacarpal joint, rendering the thumb ineffective of opposition, thereby destroying the essential element of grasp and pinch. In a study of 76 hands it was found that it was the dominant hand of the family bread earner, which was most commonly involved, and proper primary management could avoid the development of this contracture. Adequate release of the contractured first web space from hinge to hinge followed by a local skin flap cover and dynamic abduction splintage for the subsequent six weeks gives good results and prevents recurrences. Secondary surgeries like Flexor Pollicis Longus lengthening and opponensplasty are sometimes added to avoid recurrences. Despite advances in surgical technique, operative magnification, better suture materials and improved tension less suture techniques, recovery of peripheral nerve function after injury is dependent on many a factors that elude the surgeon. We are unable to alter the process of Wallerian degeneration and the devastating effect it has on the motor end plates, sensory end organs and thus the overall functional capability of the patient. The age of the patient, the level of injury, the mechanism of injury and the flexibility of the patient’s joints and psyche all determine how well a nerve regenerates while the surgeon can only wait, watch and hope. In this not so encouraging scenario, entrapment neuropathy or compression neuropathy remains a silver lining. Compression
Neuropathy refers to a group of syndromes resulting from entrapment or
compression of a short segment of a nerve at a specific site. Frequently
the nerve is vulnerable where it passes through a fibro-osseous tunnel or
an opening in fibrous or muscular tissue. Compression neuropathy refers to
nerve damage due to pressure applied to the nerve; entrapment neuropathy
applies when the pressure is exerted by some anatomic structure.
Therefore, entrapment neuropathies are chronic conditions, but compression
neuropathies may be acute. A careful history including patient’s occupation, metabolic factors, environmental exposure and social milieu is essential to come to a definitive diagnosis. It is always wise to identify the site of neuropathy and then attempt to establish its cause and any contributory associated factors. For instance, a Median neuropathy at the wrist can be from compression, injury, diabetes, hypothyroidism or perhaps repetitive movements across the carpel tunnel. Etiologies may be confusing when neuropathies are related to unsuspected systemic diseases like diabetes, hypothyroidism and gout. The
basic etiology however remains to be a newly developed disparity between
the volume of contents of a fibro-osseous / fibro-fascial tunnel and its
capacity, which in turn compresses the contained nerve. Causes can be
pressure from outside the tunnel, in the wall of the tunnel or inside
its lumen and the effect is compression, constriction or mechanical irritation
of the contained nerve. Some common nerve entrapment sites and the resulting
syndromes are listed in the following table along with hyperlinks for
detailed reading and appreciation:
Patients
complain of numbness, loss of movement, swelling, prickling, mild-to-severe
pain often worse at night, decreased sensation and decreased strength
in hand and fingers. Other symptoms may include tendency to drop things
and difficulty manipulating small objects. A careful physical examination,
Neurological
evaluation, Electromyography,
Nerve
conduction velocity test, and at times MRI,
Ultrasound and X-ray
are required to establish the diagnosis. The role of imaging is gaining
more and more ground as we encounter unexpected causes of Carpel Tunnel
Syndrome preoperatively such as a large adductor pollicis muscle, a persistent
Median artery, an excessive amount of fatty tissue within the tunnel,
a ganglion cyst and synovial hypertrophy related to rheumatoid arthritis. Treatment focuses on identifying and removing or correcting the underlying cause of the nerve compression. Some interesting treatment options like autogenous saphanous vein wrapping of the Median nerve at wrist for recurrent compressive neuropathy also find a mention. The Ulnar nerve at the elbow level is relieved of compression by either anterior transposition or medial epicondylectomy. The treatment options include vocational or occupational counseling, wrist splint and other splints, over-the-counter pain medication or prescription drugs, Surgery and Physical therapy. Taking safety measures to compensate for loss of sensation is of vital importance. The pathophysiology of Compression Neuropathy is a chronic blunt injury to the peripheral nerve resulting in microvascular ischemic changes, oedema, dislocation of the nodes of Ranvier, and structural alterations in the membranes at the organelle levels in both the myelin sheath and the axon. Focal segmental demyelination is a constant feature. Complete recovery of function after surgical decompression represents remyelination. Incomplete recovery in more chronic and severe cases is due to Wallerian degeneration of the axons and permanent fibrotic changes in the neuro-muscular junctions that prevent reinnervation. |
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